Recent research using brain scans showed iron deposition in patients with CCSVI and MS. But the iron deposits weren’t found in areas typically associated with hyperintensity signals. Instead, they were found in different but very interesting locations that I won’t go into here. Suffice it to say that sluggish brain circulation can lead to cytotoxic edema, which is a topic I cover thoroughly in my book. Sluggish blood flow causes all types of waste products to accumulate and iron is just one of several well known suspects that can initiate neurodegenerative processes. Other strong suspects include calcium and glutamate. Nonetheless, knowing all this, still doesn’t tell us what the cause of CCSVI and sluggish blood flow is. Furthermore, except for research, it serves no clinical purpose to use brain scans to check for iron deposits as a marker for MS considering the cost and the fact that it does nothing to alter the course of the condition.
What’s more interesting about this recent study on iron depositon is that it cites two other particular studies of interest. The first one from Brain Research, done in 1997, showed similar iron deposition in Alzheimer’s disease, which I cover in my book. The other study is from Functional Neurology, by Dr. Zamboni where it was found that the severity of CCSVI, that is venous drainage issues in the brain, is related to altered CSF hydrodynamics. All three studies corroborate my theory that, in addition to MS, CCSVI similarly causes Alzheimer’s and Parkinson’s diseases. I will be covering cranial hydrodynamics in future posts. I cover the topic thoroughly in my book. Among other things, I believe that CCSVI alters cranial hydrodynamics resulting in either hydrocephalus or Chiari type conditions, but it starts with drainage issues.